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When GD is treated with thionamide antithyroid drugs (ATD), hypothyroidism develops in a certain percentage of patients after several weeks to months due to the TSH receptorblocking effect of the thionamide, leading to a decrease in the titer of the newly released antibody [6, 7]
It is characterized by ROS-induced dissociation of the oxygen-sensitive kinase KEAP1, activating the NRF2 signaling pathway and caspase-8-dependent cell death [241, 242]
published online October 4
If your prescriber checks kidney function periodically, expect mild fluctuations
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